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    none: Fig. 1. Cas Hey. Région protubérantielle moyenne. Lésion bilatérale. Destruction complète calotte côté droit, incomplète au côté gauche / Fig. 2. Cas Hey. Destruction unilatérale de la calotte. Petit foyer accessoire dans la partie haut du pied au-dessus des fibres pyramidales
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    none: Fig. 3. Cas Hey. Bulbe supérieur. Dégénération du F. C. Calote. Aspect pseudo-hypertrophique de l'olive / Fig. 4 Cas Hey. Bulbe un peu plus bas. Mêmes indications que la figure précédente
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    none: Fig. 5. Cas Hey. Moelle cervicale supérieure. Atteinte bilatérale du faisceau de Helweg
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    none: Fig. 6. Cas L. Marie. Lésion unilatérale. Calotte protubérantielle moyenne. Destruction bandelette et F. C. calotte du même côté / Fig. 7. Cas L. Marie. Bulbe supérieur. Dégénération F. C. calotte. Aspect pseudo-hypertrophique olive
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    none: Fig. 8. Cas No... Dégénération F. C. calotte et léger aspect pseudo-hypertrophique olive
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    none: Fig. 9. Cas Bo... Lacunne de la calotte touchant le F. C. et respectant la bandelette long. Région protubérantielle moyenne / Fig. 10. Cas Bo... Dégénération F. C. calotte d'aspect pseudo-hypertrophique
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    none: Fig. 11. [Le problème anatomique du nystagmus du voile du palais] / Fig. 12. Cas Bo... Mêmes lésions à un plus fort grossissement montrant l'intensité de la lésion du faisceau central de la calotte
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    none: Fig. 3. Coupe transversale du nerf médian (faible grossissement) / Fig. 4. 1, nerf intercostal ; 2, nerf phrénique ; 3, nerf sympathique ; 4, nerf pneumogastrique
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    none: 0113 - Page 57 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le syndrome métastatique aigu et régressif des tumeurs secondaires du cerveau, par MM. Raymond Garcin et René Huguenin
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    none: 0114 - Page 58 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le syndrome métastatique aigu et régressif des tumeurs secondaires du cerveau, par MM. Raymond Garcin et René Huguenin
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    none: 0115 - Page 59 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le syndrome métastatique aigu et régressif des tumeurs secondaires du cerveau, par MM. Raymond Garcin et René Huguenin / Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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  none: 0116 - Page 60 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0116 - Page 60 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: Fig. 1. Le sujet à 20 ans
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  none: 0117 - Page 61 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0117 - Page 61 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: Fig. 2. Le malade à 28 ans. Facies myxoedémateux
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  none: 0118 - Page 62 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0118 - Page 62 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: Fig. 3. Aspect d'ensemble où l'on note l'hypertrophie du trapèze droit, des éminences thénar, et les boules musculaires des jumeaux
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  none: 0119 - Page 63 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0119 - Page 63 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: Fig. 4. L'hypertrophie des quadriceps et des vastes de la cuisse est de profil particulièrement nette. Attitude de repos
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  none: 0120 - Page 64 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0120 - Page 64 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: Fig. 5. Autre aspect du membre inférieur qui montre encore l'hypertrophie musculaire des quadriceps de la cuisse et des muscles jumeaux
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    none: 0121 - Page 65 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0122 - Page 66 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0123 - Page 67 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0124 - Page 68 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0125 - Page 69 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0126 - Page 70 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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    none: 0127 - Page 71 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané et évolution parallèle (premier mémoire). Etude clinique, par MM. Raymond Garcin, L. Rouques, Laudat et Frumusan
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  none: 0131 - Page 75 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: 0131 - Page 75 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: Fig. 1. (Courbe n° 6) Enregistrement du gonflement du muscle
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  none: 0132 - Page 76 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: 0132 - Page 76 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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  none: 0133 - Page 77 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: 0133 - Page 77 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: Fig. 3. (Courbe n° 17) Enregistrement des mouvements du pouce / Fig. 4. (Courbe n°3) Enregistrement des mouvements du pouce
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  none: 0134 - Page 78 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: 0134 - Page 78 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: Fig. 5. (Dernière courbe de cette série) Enregistrement des mouvements du pouce
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  none: 0135 - Page 79 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: 0135 - Page 79 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: 0136 - Page 80 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: 0137 - Page 81 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin
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    none: 0138 - Page 82 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et myxoedème cliniquement associés. Début simultané, évolution parallèle (2e mémoire). Etude de la myotonie, par MM. Georges Bourguignon et Raymond Garcin / Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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  none: 0139 - Page 83 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: 0139 - Page 83 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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  none: 0140 - Page 84 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: 0140 - Page 84 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: Fig. 1. Coupe transversale du muscle / Fig. 2. Coupe longitudinale du muscle
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  none: 0141 - Page 85 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: 0141 - Page 85 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: Fig. 3. Coupe longitudinale du muscle / Fig. 4. Aspect méconnaissable de la glande thyroïde-scléreuse et très atrophiée
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  none: 0142 - Page 86 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: 0142 - Page 86 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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  none: 0143 - Page 87 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: 0143 - Page 87 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: Fig. 5. Coupe du testicule
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    none: 0144 - Page 88 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: 0145 - Page 89 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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  none: 0146 - Page 90 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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    none: 0146 - Page 90 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Syndrome thomsénien et syndrome myxoedémateux cliniquement associés. Début simultané. Evolution parallèle (3e mémoire). Etude anatomique et conclusions, par MM. Raymond Garcin et Ivan Bertrand
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  none: 0147 - Page 91 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: 0147 - Page 91 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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  none: 0148 - Page 92 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: 0148 - Page 92 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: Fig. 1. Cas I. M. Section transversale de la moelle dorsale
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  none: 0149 - Page 93 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: 0149 - Page 93 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: Fig. 2. Cas I. M. Deux segments de la moelle dorsale (coloration myélinique) / Fig. 3. Cas I. M. Région dorsale inférieure
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  none: 0150 - Page 94 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: 0150 - Page 94 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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  none: 0151 - Page 95 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: 0151 - Page 95 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: Fig. 4. Cas III. J. (Coloration myélinique). a C4 : cavité syringomyélique à son maximum de développement ; épaississement du ligament postérieur. b. D3 : La cavité syringomyélique est plus petite ; le feutrage arachnoïdien est plus dense et occupe tout l'espace postérieur. La moelle ne présente aucune dégénération myélinique marginale / Fig. 5. Cas VI. Elargissement du ligament postérieur de la moelle
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  none: 0152 - Page 96 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: 0152 - Page 96 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: 0153 - Page 97 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André
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    none: 0154 - Page 98 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le feutrage arachnoïdien postérieur dans la syringomyélie (sa place dans la pathologie des leptoméninges), par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. André / Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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  none: 0155 - Page 99 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: 0155 - Page 99 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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  none: 0156 - Page 100 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: 0156 - Page 100 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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  none: 0157 - Page 101 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: 0157 - Page 101 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: Fig. 1. (coloration myélinique : Kultschitzky-Pal + cochenille) ; a C6, démyélinisations circonscrites, analogiques à la sclérose en plaques ; b C8, même type de lésion ; c D (en bas), myélite diffuse. Phénomènes inflammatoires dans les cordons postérieurs. Démyélinisation très étendue mais diffuse
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  none: 0158 - Page 102 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: 0158 - Page 102 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: Fig. 2. (col. Nissl). Aspect aréolaire de la moelle au niveau de C8 / Fig. 3. (col. van Gieson). Détail des lésions de myélite diffuse (moelle dorsale)
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  none: 0159 - Page 103 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: 0159 - Page 103 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: Fig. 4. Bulbe (col. myélinique spielmeyer à congélation). Démyélinisation circonscrite
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    none: 0160 - Page 104 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: Fig. 5. N. en haut (coloration Nissl). Aspect caractéristique en trois zones des foyers cérébraux. M. en bas : coloration myélinique. Ce foyer présente des caractères particuliers
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    none: 0161 - Page 105 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas anatomo-clinique de sclérose en plaques aiguë avec symptomatologie de neuropticomyélite, par MM. Th. Alajouanine, Th. Hornet, R. Thurel et R. Rossano
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    none: Fig. 6. En haut, chiasma optique (coloration myélinique) ; en bas, nerf optique droit
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    none: Fig. 1. Anomalie structurale de l'écorce cérébrale / Fig. 2. Calcifications en sphérules dans la partie fibrillaire de la tumeur (éosine hématoxyline)
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    none: 0169 - Page 113 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas de sclérose tubéreuse avec spongioblastome paraventriculaire, par MM. J. Lhermitte, Heuyer et Mme Claire Vogt
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    none: Fig. 3. Partie fibreuse de la tumeur avec infiltrations calcaires
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    none: 0170 - Page 114 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Un cas de sclérose tubéreuse avec spongioblastome paraventriculaire, par MM. J. Lhermitte, Heuyer et Mme Claire Vogt
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    none: Fig. 4. Partie cellulaire de la tumeur avec nombreux éléments monstrueux, quelques blocs calcifiés (Eosine hématoxyline)
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    none: 0171 - Page 115 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Polioencéphalomyélite intermittente ou myasthénie (Episodes paralytiques successifs répartis sur une durée de 27 ans avec, actuellement, apparition d'un syndrome myasthénique), par MM. Th. Alajouanine, R. Thurel et R. André
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    none: 0172 - Page 116 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Polioencéphalomyélite intermittente ou myasthénie (Episodes paralytiques successifs répartis sur une durée de 27 ans avec, actuellement, apparition d'un syndrome myasthénique), par MM. Th. Alajouanine, R. Thurel et R. André
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    none: 0174 - Page 118 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Ramollissement cérébral à foyer double symétrique consécutif à la ligature d'une veine jugulaire, par MM. Pierre Duval, J. Lhermitte et Vermes
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  none: 0175 - Page 119 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Ramollissement cérébral à foyer double symétrique consécutif à la ligature d'une veine jugulaire, par MM. Pierre Duval, J. Lhermitte et Vermes
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    none: 0175 - Page 119 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Ramollissement cérébral à foyer double symétrique consécutif à la ligature d'une veine jugulaire, par MM. Pierre Duval, J. Lhermitte et Vermes
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    none: Fig. 1. Ramollissement hémorragique du lobe frontal et des circ. rolandiques gauches
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    none: 0176 - Page 120 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Ramollissement cérébral à foyer double symétrique consécutif à la ligature d'une veine jugulaire, par MM. Pierre Duval, J. Lhermitte et Vermes
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    none: Fig. 2. Nécrose du tissu nerveux et nombreux hémorragies de l'écorce rolandique
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    none: 0177 - Page 121 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Ramollissement cérébral à foyer double symétrique consécutif à la ligature d'une veine jugulaire, par MM. Pierre Duval, J. Lhermitte et Vermes
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    none: Fig. 3. Dévastation de l'écorce cérébrale / Fig. 4. Cytolyse des cellules nerveuses, prolifération des microgliocytes, diapédèse pariéto-vasculaire dans une région libre de toute hémorragie. Niecks de Nissl
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    none: 0180 - Page 124 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Ramollissement cérébral à foyer double symétrique consécutif à la ligature d'une veine jugulaire, par MM. Pierre Duval, J. Lhermitte et Vermes
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    none: 0181 - Page 125 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Ramollissement cérébral à foyer double symétrique consécutif à la ligature d'une veine jugulaire, par MM. Pierre Duval, J. Lhermitte et Vermes / Diabète insipide en apparence consécutif à une entorse tibio-tarsienne. Les facteurs étiologiques lointains et immédiats de la polyurie, par MM. J. Lhermitte, Metenier et Gauthier
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    none: 0182 - Page 126 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Diabète insipide en apparence consécutif à une entorse tibio-tarsienne. Les facteurs étiologiques lointains et immédiats de la polyurie, par MM. J. Lhermitte, Metenier et Gauthier
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    none: 0183 - Page 127 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Diabète insipide en apparence consécutif à une entorse tibio-tarsienne. Les facteurs étiologiques lointains et immédiats de la polyurie, par MM. J. Lhermitte, Metenier et Gauthier / La pachybasie sellaire et sa valeur séméiologique, par MM. J. Lhermitte et Nemours-Auguste
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  none: 0184 - Page 128 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La pachybasie sellaire et sa valeur séméiologique, par MM. J. Lhermitte et Nemours-Auguste
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    none: 0184 - Page 128 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La pachybasie sellaire et sa valeur séméiologique, par MM. J. Lhermitte et Nemours-Auguste
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  none: 0185 - Page 129 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La pachybasie sellaire et sa valeur séméiologique, par MM. J. Lhermitte et Nemours-Auguste
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    none: 0185 - Page 129 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La pachybasie sellaire et sa valeur séméiologique, par MM. J. Lhermitte et Nemours-Auguste
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    none: Fig. 1. M..., 48 ans. Remarquer l'énorme massif sellaire, l'hypertrophie des clinoïdes, le faible développement du sinus sphénoïdal
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    none: 0186 - Page 130 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La pachybasie sellaire et sa valeur séméiologique, par MM. J. Lhermitte et Nemours-Auguste / Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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  none: 0187 - Page 131 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: 0187 - Page 131 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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  none: 0188 - Page 132 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: 0188 - Page 132 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: Fig. 1. Coupe horizontale montrant l'augmentation de volume de l'hémisphère droit, la déviation de la scissure interhémispherique, le tubercule frontal et le ramollissement du centre ovale à droite de la capsule externe et interne
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  none: 0189 - Page 133 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: 0189 - Page 133 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: Fig. 2. Coupe horizontale par la tête du noyau caudé (Loyez)
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  none: 0190 - Page 134 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: 0190 - Page 134 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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  none: 0191 - Page 135 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: 0191 - Page 135 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: Fig. 3. Zone méso-diencéphalique des centres régulateurs du sommeil d'après von Economo (en pointillé)
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  none: 0192 - Page 136 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: 0192 - Page 136 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: Fig. 4. Foyer d'épendymite à la partie supérieure de l'aqueduc (Prolifération épithéliale et névroglique. Oedème sous-épithélial)
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  none: 0193 - Page 137 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: 0193 - Page 137 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Contribution histopathologique à l'étude de la narcolepsie et du tremblement avec rigidité musculaire. Tubercule du lobe frontal avec lésions diencéphaliques juxtaventriculaires et lésions des corps striés, par M. Marcel Monnier (présenté par M. Guillain)
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    none: Fig. 5. Proliférations épendymaires papilliformes à partir de la paroi ventriculaire droite, près du plancher dans la région tubérienne
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    none: Fig. 6. Saillie épendymaire dans le 3e ventricule à partir de la paroi ventriculaire droite, au-dessus de la commissure blanche postérieure
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    none: 0202 - Page 146 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Discordance du lipiodiagnostic sous-arachnoïdien et de la manoeuvre de Queckenstedt dans un cas de tumeur médullaire, par MM. F. Coste et J. Haguenau
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    none: 0203 - Page 147 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Discordance du lipiodiagnostic sous-arachnoïdien et de la manoeuvre de Queckenstedt dans un cas de tumeur médullaire, par MM. F. Coste et J. Haguenau
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    none: Fig. 1. Calque radiographique de Mme T... Lipiodiagnostic de bas en haut
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    none: 0204 - Page 148 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Discordance du lipiodiagnostic sous-arachnoïdien et de la manoeuvre de Queckenstedt dans un cas de tumeur médullaire, par MM. F. Coste et J. Haguenau
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    none: Fig. 2. Dessin montrant le siège de la tumeur, l'implantation latérale, les racines postérieures accolées qui la cravatent
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    none: 0205 - Page 149 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Discordance du lipiodiagnostic sous-arachnoïdien et de la manoeuvre de Queckenstedt dans un cas de tumeur médullaire, par MM. F. Coste et J. Haguenau / La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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  none: 0206 - Page 150 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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    none: 0206 - Page 150 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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  none: 0207 - Page 151 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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    none: 0207 - Page 151 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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    none: Fig. 1. Tumeur fronto-temporale droite ayant refoulé le ventricule latéral et le 3e ventricule à gauche / Fig. 2. Cas 1, corne frontale droite dilatée et débordant la ligne médiane du côté opposé
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  none: 0208 - Page 152 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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    none: 0208 - Page 152 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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    none: Fig. 3. Cas 1, corne frontale gauche dilatée et débordant légèrement la ligne médiane
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  none: 0209 - Page 153 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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    none: 0209 - Page 153 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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    none: Fig. 4. Schéma des ventricules montrant en trait plein les limites des ventricules à l'état normal
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  none: 0210 - Page 154 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. La ventriculographie dans les cas d'imperméabilité du trou de Monro, par M. Léon Ectors
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  none: 0214 - Page 158 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Sclérose latérale amyotrophique de type poliomyliétique à début bulbaire, par M. Léon Ectors
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    none: 0214 - Page 158 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Sclérose latérale amyotrophique de type poliomyliétique à début bulbaire, par M. Léon Ectors
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    none: 0215 - Page 159 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Sclérose latérale amyotrophique de type poliomyliétique à début bulbaire, par M. Léon Ectors
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    none: Fig. 1. Nissl. Noyau du grand hypoglosse montrant la dégénérescence des neurones à des stades divers / Fig. 2. Nissl. Aspect d'ensemble de la corne antérieure dorsale
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    none: 0216 - Page 160 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Sclérose latérale amyotrophique de type poliomyliétique à début bulbaire, par M. Léon Ectors
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    none: Fig. 3. Nissl. Réaction périvasculaire d'intensité moyenne au niveau du faisceau pyramidal croisé au niveau de la moelle dorsale
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    none: 0217 - Page 161 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Sclérose latérale amyotrophique de type poliomyliétique à début bulbaire, par M. Léon Ectors
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    none: Fig. 4. Weigert. Remarquer l'atrophie marquée des cornes antérieures et l'absence de dégénérescence myélinique visible
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    none: 0218 - Page 162 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Sclérose latérale amyotrophique de type poliomyliétique à début bulbaire, par M. Léon Ectors
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    none: 0219 - Page 163 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Sclérose latérale amyotrophique de type poliomyliétique à début bulbaire, par M. Léon Ectors (Travail de la Clinique neurologique de la Salpêtrière du Pr Guillain) / Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: 0221 - Page 165 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: 0222 - Page 166 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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  none: 0223 - Page 167 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: 0223 - Page 167 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: Fig. 1. Réseau fibrillaire du système végétatif périventriculaire (zone de l'aqueduc de Sylvius) (chien)
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  none: 0224 - Page 168 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: 0224 - Page 168 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: Fig. 2. Réseau fibraillaire sous-épendymaire dans la zone protubérantielle (chien)
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  none: 0225 - Page 169 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: 0225 - Page 169 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: Fig. 3. Réseau épendymaire au niveau du IIIe ventricule dont la cavité est à droite (hypothalamu antérieur de l'homme)
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    none: 0226 - Page 170 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: 0227 - Page 171 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Le système neuro-végétatif périventriculaire ou sous-épendymaire, par MM. G. Roussy et M. Mosinger
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    none: Schéma. 1. Le système neurovégétatif périventriculaire sousépendymaire
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    none: 0246 - Page 190 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Deux cas d'hémorragie musculaire consécutive à une intoxication alcoolique suivie de causalgie. Traitement par l'hyposulfite de sodium, par M. Smirnow
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    none: 0247 - Page 191 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Deux cas d'hémorragie musculaire consécutive à une intoxication alcoolique suivie de causalgie. Traitement par l'hyposulfite de sodium, par M. Smirnow / Compression médullaire par épidurite chronique staphylococcique chez une diabétique, par MM. Raymond Garcin, Petit-Dutaillis et Ivan Bertrand
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  none: 0248 - Page 192 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Compression médullaire par épidurite chronique staphylococcique chez une diabétique, par MM. Raymond Garcin, Petit-Dutaillis et Ivan Bertrand
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    none: 0248 - Page 192 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Compression médullaire par épidurite chronique staphylococcique chez une diabétique, par MM. Raymond Garcin, Petit-Dutaillis et Ivan Bertrand
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    none: 0249 - Page 193 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Compression médullaire par épidurite chronique staphylococcique chez une diabétique, par MM. Raymond Garcin, Petit-Dutaillis et Ivan Bertrand
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    none: Fig. 1. Noter l'aplatissement de la moelle et l'épaississement de la méninge dure qui fait corps en arrière avec le fuseau d'épidurite (non représenté ici) étendu sur une dizaine de centimètres de hauteur
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  none: 0250 - Page 194 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Compression médullaire par épidurite chronique staphylococcique chez une diabétique, par MM. Raymond Garcin, Petit-Dutaillis et Ivan Bertrand
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    none: 0250 - Page 194 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Compression médullaire par épidurite chronique staphylococcique chez une diabétique, par MM. Raymond Garcin, Petit-Dutaillis et Ivan Bertrand
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    none: Fig. 2. Texture de la dure-mère au voisinage de la gangue épidurale qui l'environne. Coupe longitudinale
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    none: 0251 - Page 195 - Société de neurologie de Paris. Séance du 4 juillet 1935. Communications. Compression médullaire par épidurite chronique staphylococcique chez une diabétique, par MM. Raymond Garcin, Petit-Dutaillis et Ivan Bertrand
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